Loading ...Published on May 23, 2020
How did this Common Cold virus from the barnyard turn into something infectious in people?
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How did this Animal Virus become a Human Virus? Back in the 1990’s, researchers at UNC (Univ of N. Carolina) mentored exchange students from WIV (Wuhan Institute of Virology) on how to breed virulent viruses in their ‘CoV Pathogenesis Program’ (pathogenesis: “to cause disease”). By persistent passages of virus through monkey kidney cells, CoV learned how to enter epithelial cells through ACE-2 receptors, something ubiquitous throughout our “inner skin” that runs the plumbing for everything. But this virus needed to be passaged through its target human host (down in the Guangzhou ghetto bordering Hong Kong), and needed to have its genes recombined into a new hybrid species through ‘Gain-Of-Function’ techniques using the result from human passage.
This new CoV virus was called a ‘Recombinant’, from combining genes from different strains, and it needed to be passaged through its target host, again – to make a strain virulent enough for a vaccine. Weak viruses, the ones that have achieved tolerance in their hosts, do not make good candidates for immune stimulants. You can’t make a vaccine without a good pathogenic candidate.